Schizophrenia typically begins:
a) In early childhood
b) During late teens or early adulthood
c) After age 50
d) Only in people with Alzheimer’s
During late teens or early adulthood
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| Term | Definition |
|---|---|
| Schizophrenia typically begins: a) In early childhood b) During late teens or early adulthood c) After age 50 d) Only in people with Alzheimer’s | During late teens or early adulthood |
| Which genes are associated with schizophrenia? a) C4, DRD2, and glutamate-related genes b) APP and APOE ε4 c) PSEN1 and PSEN2 d) Serotonin transporter (SERT) only | C4, DRD2, and glutamate related |
| The C4 gene in schizophrenia is involved in: a) Memory storage b) Immune system and synapse pruning c) Hormone regulation d) Muscle contraction | Immune system and synapse pruning |
| Alzheimer’s disease is characterized by: a) Amyloid plaques and tau tangles b) Increased dopamine production c) Overactive telomerase d) Loss of serotonin receptors | Amyloid plaques and tau tangles |
| Which genes are linked to Alzheimer’s disease? a) C4, DRD2 b) APP, PSEN1, PSEN2, and APOE ε4 c) 5-HTT and MAOA d) None — it’s purely environmental | APP, PSEN1, PSEN2, and APOE ε4 |
| The APOE ε4 allele is associated with: a) Reduced risk of heart disease b) Increased risk of Alzheimer’s c) Stronger immune function d) Faster metabolism | Increased risk of Alzheimer’s |
| Depression has been linked to: a) Chromosome 3 and low serotonin levels b) Chromosome 21 and high dopamine levels c) Amyloid buildup d) C4 and DRD2 | Chromosome 3 and low serotonin levels |
| Low levels of serotonin are most closely associated with: a) Schizophrenia b) Alzheimer’s disease c) Depression d) Telomere shortening | Depression |
| Genetic aging refers to: a) Random damage over time b) A programmed lifespan encoded in genes c) Environmental wear and tear d) Cellular damage caused by UV light | A programmed lifespan encoded in genes |
| Physiological aging results from: a) Mutations only b) Wear, damage, and environmental stress c) Genetic programming d) Overactive telomerase | Wear, damage, and environmental stress |
| Senescence occurs when: a) Cells die b) Cells stop dividing but remain metabolically active c) DNA replication speeds up d) Cells become cancerous | Cells stop dividing but remain metabolically active |
| Telomeres are: a) Proteins that digest waste b) Caps on chromosomes that shorten with each division c) Immune cells that prevent aging d) Enzymes that repair mutations | Caps on chromosomes that shorten with each division |
| The enzyme telomerase: a) Speeds up aging b) Repairs damaged proteins c) Rebuilds telomeres and may slow aging d) Increases oxidative stress | Rebuilds telomeres and may slow aging |
| Accumulation of mutations contributes to aging by: a) Preventing cell death b) Damaging DNA and cellular processes c) Increasing telomerase activity d) Boosting metabolism | Damaging DNA and cellular processes |
| Humans tend to age faster after about age 60 because: a) Natural selection weakens with age b) Telomeres lengthen c) The immune system strengthens d) Mutation rates drop | Natural selection weakens with age |
| Calorie restriction, senolytics, and telomerase therapy are examples of: a) Cancer treatments b) Anti-aging research strategies c) Gene editing tools d) Mental health therapies | Anti-aging research strategies |